CANADIAN JOURNAL OF CARDIOLOGY, vol.27, no.6, pp.773-778, 2011 (SCI-Expanded)
Background: The pathophysiology of coronary artery ectasia (CAE) is still unknown. Inflammation and degradation of connective tissue may have a role in the development of coronary ectasia. In the present study, the authors examined neutrophil gelatinase-associated lipocalin (NGAL) levels in isolated CAE patients.